Table Legends
Table 1. Characteristics of study subjects. EC,
Endemic control; LTBi, latent TB; DM, diabetes mellitus; DM-LTBi,
latently infected Diabetics; TB, tuberculosis; RBS, random blood sugar.
Table 2. Differential pro-inflammatory cytokine secretion
between healthy controls, diabetics and LTBi cases. EC, Endemic
control; LTBi, latent TB; DM, diabetes mellitus; DM-LTBi, latently
infected Diabetics; TB, tuberculosis. Supernatants were collected and
tested for IFN-γ, IL-12p70, IL-2, TNF-α, GM-CSF, and IL-1β by Th1 Th2 11
plex procarta plex assay (Thermofischer, USA). Data is depicted as
median with IQR. Kruskal Wallis test was run between the groups to see
the difference in secretion levels and P values are included in the
table. Individual groups were also compared using Mann–Whitney U-test.
*Denotes a significant difference (≤0.05) as compared with DM-LTBi; #
as compared with EC; NS, not significantly different,
p>0.05.
Table 3. Differential type 2 cytokine secretion between
healthy controls, diabetics and LTBi cases. EC, Endemic control; LTBi,
latent TB; DM, diabetes mellitus; DM-LTBi, latently infected diabetics;
TB, tuberculosis. Supernatants were collected and tested for IL-13,
IL-4, IL-5, IL-6 and IL-18 by Th1 Th2 11 plex procarta plex assay
(Thermofischer, USA). Data is depicted as median with IQR. Kruskal
Wallis test was run between the groups to see the difference in
secretion levels and P values are included in the table. Individual
groups were also compared using Mann–Whitney U-test. *Denotes a
significant difference (≤0.05) as compared with DM-LTBi; NS, not
significantly different, p>0.05.